https://ogma.newcastle.edu.au/vital/access/ /manager/Index en-au 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:12695 Wed 24 Jul 2013 22:25:15 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:21456 Tue 24 Aug 2021 14:35:44 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:7708 Sat 24 Mar 2018 08:41:37 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:8031 Sat 24 Mar 2018 08:36:47 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:12710 A) was maintained sequentially at 22°C, 12°C, 22°C and 35°C, with an accuracy of ±1°C. Neither thalamic nor pontine rabbits could maintain core temperature in cold or heat. At T 35°C, thalamic and pontine animals did not pant, indicating that telencephalic responses were necessary for the integration of mechanisms promoting respiratory heat loss. Thalamic animals, however, could inhibit ear vascular sympathetic tone in the heat, but the response was absent in pontine animals, suggesting diencephalic responses were essential for the integration of mechanisms promoting ear skin heat loss. Thus, the neural adjustments to thermal stress depend on mechanisms of integration distributed longitudinally throughout the central nervous sytem, and different components of the reflex cardiorespiratory response depend on different sites in the central nervous system for their full expression.]]> Sat 24 Mar 2018 08:16:20 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:16912 A receptor antagonist) into sites within a circumscribed region in the deep layers of the superior colliculus and in the central and external nuclei of the inferior colliculus evoked a response characterized by intense and highly synchronized bursts of renal sympathetic nerve activity (RSNA) and phrenic nerve activity (PNA). Each burst of RSNA had a duration of ~300–400 ms and occurred slightly later (peak to peak latency of 41 ± 8 ms) than the corresponding burst of PNA. The bursts of RSNA and PNA were also accompanied by transient increases in arterial pressure and, in most cases, heart rate. Synchronized bursts of RSNA and PNA were also evoked after neuromuscular blockade, artificial ventilation, and vagotomy and so were not dependent on afferent feedback from the lungs. We propose that the synchronized sympathetic-respiratory responses are driven by a common population of neurons, which may normally be activated by an acute threatening stimulus.]]> Sat 24 Mar 2018 07:59:52 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:31005 Mon 23 Sep 2019 10:08:51 AEST ]]>